Detalhe da pesquisa
1.
p66Shc signaling is involved in stress responses elicited by anthracycline treatment of rat cardiomyoblasts.
Arch Toxicol
; 90(7): 1669-84, 2016 Jul.
Artigo
em Inglês
| MEDLINE | ID: mdl-26318906
2.
Mitochondrial apoptosis-inducing factor is involved in doxorubicin-induced toxicity on H9c2 cardiomyoblasts.
Biochim Biophys Acta
; 1842(12 Pt A): 2468-78, 2014 Dec.
Artigo
em Inglês
| MEDLINE | ID: mdl-25283819
3.
The Enhanced Efficacy of Intracellular Delivery of Doxorubicin/C6-Ceramide Combination Mediated by the F3 Peptide/Nucleolin System Is Supported by the Downregulation of the PI3K/Akt Pathway.
Cancers (Basel)
; 13(12)2021 Jun 18.
Artigo
em Inglês
| MEDLINE | ID: mdl-34207464
4.
Mitochondrial disruption occurs downstream from ß-adrenergic overactivation by isoproterenol in differentiated, but not undifferentiated H9c2 cardiomyoblasts: differential activation of stress and survival pathways.
Int J Biochem Cell Biol
; 45(11): 2379-91, 2013 Nov.
Artigo
em Inglês
| MEDLINE | ID: mdl-23958426
5.
Differentiation-dependent doxorubicin toxicity on H9c2 cardiomyoblasts.
Cardiovasc Toxicol
; 12(4): 326-40, 2012 Dec.
Artigo
em Inglês
| MEDLINE | ID: mdl-22744233